Araştırma Makalesi
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Yıl 2025, Cilt: 52 Sayı: 1, 7 - 16, 14.03.2025
https://doi.org/10.5798/dicletip.1657092

Öz

Kaynakça

  • 1.Altın H, Karataş Z, Şap F, et al. The associationbetween pulmonary arterial hypertension andmalnutrition in children with congenital heartdiseases with left-to-right shunt: an observationalstudy. Anadolu Kardiyol Derg 2012; 12:150-9.
  • 2.Okoromah CA, Ekure EN, Lesi FE, et.al. Prevalence,profi le and predictors of malnutrition in childrenwith congenital heart defects: a case–controlobservational study. Arch Dis Child 2011; 96:354-60.
  • 3.Varan B, Tokel K, Yılmaz G. Malnutrition andgrowth failure in cyanotic and acyanotic congenitalheart disease with and without pulmonaryhypertension. Arch Dis Child 1999; 81:49-52.
  • 4.Medoff-Cooper B, Ravishankar C. Nutrition andgrowth in congenital heart disease: a challenge inchildren. Curr Opin Cardiol 2013, 28:122-9.
  • 5.Vaidyanathan B, Radhakrishnan R, Sarala DA,Sundaram KR, Kumar RK. What determinesnutritional recovery in malnourished children aftercorrection of congenital heart defects? Pediatrics2009;124:e294-9.
  • 6.Arodiwe I, Chinawa J, Ujunwa F, Adiele D, UkohaM, Obidike E. Nutritional status of congenital heartdisease (CHD) patients: Burden and determinant ofmalnutrition at university of Nigeria teachinghospital Ituku–Ozalla, Enugu. Pak J Med Sci 2015;31:1140-5.
  • 7.Kyle UG, Shekerdemian LS, Coss-Bu JA. GrowthFailure and Nutrition Considerations in ChronicChildhood Wasting Diseases. Nutr Clin Pract. 2015;30:227-38.
  • 8.Irving SY, Simone SD, Hicks FW, Verger JT.Nutrition for the critically ill child: enteral and parenteral support. AACN Clin Issues 2000; 11:541-8.
  • 9.Dinleyici EC, Kılıc Z, Buyukkaragoz B, et al. SerumIGF-1, IGFBP-3 and growth hormone levels inchildren with congenital heart disease: relationshipwith nutritional status, cyanosis and left ventricularfunctions. Neuro Endocrinol Lett 2007; 28:279-83.
  • 10.Vaidyanathan B, Nair SB, Sundaram KR, et al.Malnutrition in children with congenital heartdisease (CHD) determinants and short-term impactof corrective intervention. Indian Pediatr 2008;45:541-6.
  • 11.Janes MD, Antia AU. Physical growth of childrenwith congenital heart malformations. Niger JPaediatr 1970;2 :1-8.
  • 12.Mehrizi A, Drash A. Growth disturbance incongenital heart disease. J Pediatr 1962; 61:418-29.
  • 13.Cooper BM, Maryam N, Deborah T, Antonia M.Feeding, growth and nutrition in children withcongenital malformed hearts. Cardiology in theyoung 2010;149-53.
  • 14.Pillo-Blocka F, Adatia I, Shareef W, McCrindleBW, Zlotkin S. Rapid advancement to moreconcentrated formula in infants after surgery forcongenital heart disease reduces duration ofhospital stay: a randomized clinical trial. J Pediatr2004; 145:761-6.
  • 15.Cameron JW, Rosenthal A, Olson AD.Malnutrition in hospitalized children withcongenital heart disease. Arch Pediatr Adolesc Med1995; 149: 1098-102.
  • 16.Dundar B, Akcoral A, Saylam G, et al. Chronichypoxemia leads to reduced serum IGF-I levels incyanotic congenital heart disease. J PediatrEndocrinol Metab 2000; 13: 431-6.
  • 17. Samadi M, Rashid RJ, Ghaffari S, Shoaran M. Studyon bone age in pediatric patients with congenitalheart disease and its relation with cyanosis and pulmonary artery pressure. Pak J Biol Sci 2009; 12: 702-6.
  • 18.Villasís-Keever MA, Aquiles Pineda-Cruz R,Halley-Castillo E, Alva-Espinosa C. Frecuencia yfactores de riesgo asociados a desnutrición de niñoscon cardiopatía congénita [Frequency and riskfactors associated with malnutrition in childrenwith congenital cardiopathy]. Salud Publica Mex.2001; 43:313-23.
  • 19.Menon G, Poskitt EM. Why does congenital heartdisease cause failure to thrive? Arch Dis Child 1985;60: 1134-9.
  • 20.Nydegger A, Bines JE. Energy metabolism ininfants with congenital heart disease. Nutrition2006; 22: 697-704.
  • 21.Rhee EK, Evangelista JK, Nigrin DJ, Erickson LC.Impact of anatomic closure on somatic growthamong small, asymptomatic children withsecundum atrial septal defect. Am J Cardiol 2000;85:1472-5.
  • 22.Çil E, Zincirci M, Bostan OM. Konjenital kalphastalıklarında malnutrisyon prevalansı ve bunuetkileyen faktorler. Turkiye Klinikleri Kardiyoloji2000; 13: 426-33.
  • 23.Cheung MM, Davis AM, Wilkinson JL, WeintraubRG. Long term somatic growth after repair oftetralogy of Fallot: evidence for restoration ofgenetic growth potential. Heart. 2003; 89:1340-3.
  • 24.McElhinney DB, Hedrick HL, Bush DM, et al.Necrotizing enterocolitis in neonates withcongenital heart disease: risk factors and outcomes.Pediatrics 2000; 106:1080-7.
  • 25.Jeffries HE, Wells WJ, Stames VA, Wetzel RC,Moromisato DY. Gastrointestinal morbidity afterNorwood palliation for hypoplastic left heartsyndrome. Ann Thorac Surg 2006; 81:982-7.

Prevalence of malnutrition and influencing factors in children with congenital heart disease

Yıl 2025, Cilt: 52 Sayı: 1, 7 - 16, 14.03.2025
https://doi.org/10.5798/dicletip.1657092

Öz

Background/Aim: Malnutrition is associated with increased mortality and morbidity in children with congenital heart disease (CHD). This study aims to demonstrate the factors that increase the incidence of malnutrition and to emphasize the importance of early intervention in patients with congenital heart disease.
Methods: A total of 215 patients with CHD and 242 healthy children admitted to Dicle Universty Pediatric Cardiology Clinic between November 2017 and May 2018 were included in this study. Patients with additional system diseases, dysmorphic appearance or known genetic anomalies, and premature birth or intrauterine growth retardation were excluded from the study. Nutritional statuses of the patients were questioned. The patients were evaluated by echocardiographic and anthropometric data.
Results: Malnutrition was detected in 92 patients in the patient group (42%) and 24 patients in the control group (10%); the difference was statistically significant (p<0.001). Malnutrition was detected in 26 of the 38 cyanotic patients (68.4%), in 66 of the 177 patients without cyanosis (37.2%). The rate of malnutrition was significantly higher among cyanotic patients compared to patients without cyanosis (p<0.001) and compared to the control group (p<0.001). Malnutrition was detected in 16 of the 21 patients with pulmonary hypertension (76%), and among 76 of the 194 patients without pulmonary hypertension (39%) (p<0.001). There was no significant decrease in the rate of malnutrition in patients who underwent surgical treatment compared to the patient group without surgical treatment (p=0.129). No significant increase in malnutrition rate was found in patients with CHD with left to right shunt (p: 0.190)
Conclusion: The rate of malnutrition was significantly higher among children with CHD who had pulmonary hypertension and cyanosis. There was no relationship between corrective surgery, shunting, and malnutrition development.

Kaynakça

  • 1.Altın H, Karataş Z, Şap F, et al. The associationbetween pulmonary arterial hypertension andmalnutrition in children with congenital heartdiseases with left-to-right shunt: an observationalstudy. Anadolu Kardiyol Derg 2012; 12:150-9.
  • 2.Okoromah CA, Ekure EN, Lesi FE, et.al. Prevalence,profi le and predictors of malnutrition in childrenwith congenital heart defects: a case–controlobservational study. Arch Dis Child 2011; 96:354-60.
  • 3.Varan B, Tokel K, Yılmaz G. Malnutrition andgrowth failure in cyanotic and acyanotic congenitalheart disease with and without pulmonaryhypertension. Arch Dis Child 1999; 81:49-52.
  • 4.Medoff-Cooper B, Ravishankar C. Nutrition andgrowth in congenital heart disease: a challenge inchildren. Curr Opin Cardiol 2013, 28:122-9.
  • 5.Vaidyanathan B, Radhakrishnan R, Sarala DA,Sundaram KR, Kumar RK. What determinesnutritional recovery in malnourished children aftercorrection of congenital heart defects? Pediatrics2009;124:e294-9.
  • 6.Arodiwe I, Chinawa J, Ujunwa F, Adiele D, UkohaM, Obidike E. Nutritional status of congenital heartdisease (CHD) patients: Burden and determinant ofmalnutrition at university of Nigeria teachinghospital Ituku–Ozalla, Enugu. Pak J Med Sci 2015;31:1140-5.
  • 7.Kyle UG, Shekerdemian LS, Coss-Bu JA. GrowthFailure and Nutrition Considerations in ChronicChildhood Wasting Diseases. Nutr Clin Pract. 2015;30:227-38.
  • 8.Irving SY, Simone SD, Hicks FW, Verger JT.Nutrition for the critically ill child: enteral and parenteral support. AACN Clin Issues 2000; 11:541-8.
  • 9.Dinleyici EC, Kılıc Z, Buyukkaragoz B, et al. SerumIGF-1, IGFBP-3 and growth hormone levels inchildren with congenital heart disease: relationshipwith nutritional status, cyanosis and left ventricularfunctions. Neuro Endocrinol Lett 2007; 28:279-83.
  • 10.Vaidyanathan B, Nair SB, Sundaram KR, et al.Malnutrition in children with congenital heartdisease (CHD) determinants and short-term impactof corrective intervention. Indian Pediatr 2008;45:541-6.
  • 11.Janes MD, Antia AU. Physical growth of childrenwith congenital heart malformations. Niger JPaediatr 1970;2 :1-8.
  • 12.Mehrizi A, Drash A. Growth disturbance incongenital heart disease. J Pediatr 1962; 61:418-29.
  • 13.Cooper BM, Maryam N, Deborah T, Antonia M.Feeding, growth and nutrition in children withcongenital malformed hearts. Cardiology in theyoung 2010;149-53.
  • 14.Pillo-Blocka F, Adatia I, Shareef W, McCrindleBW, Zlotkin S. Rapid advancement to moreconcentrated formula in infants after surgery forcongenital heart disease reduces duration ofhospital stay: a randomized clinical trial. J Pediatr2004; 145:761-6.
  • 15.Cameron JW, Rosenthal A, Olson AD.Malnutrition in hospitalized children withcongenital heart disease. Arch Pediatr Adolesc Med1995; 149: 1098-102.
  • 16.Dundar B, Akcoral A, Saylam G, et al. Chronichypoxemia leads to reduced serum IGF-I levels incyanotic congenital heart disease. J PediatrEndocrinol Metab 2000; 13: 431-6.
  • 17. Samadi M, Rashid RJ, Ghaffari S, Shoaran M. Studyon bone age in pediatric patients with congenitalheart disease and its relation with cyanosis and pulmonary artery pressure. Pak J Biol Sci 2009; 12: 702-6.
  • 18.Villasís-Keever MA, Aquiles Pineda-Cruz R,Halley-Castillo E, Alva-Espinosa C. Frecuencia yfactores de riesgo asociados a desnutrición de niñoscon cardiopatía congénita [Frequency and riskfactors associated with malnutrition in childrenwith congenital cardiopathy]. Salud Publica Mex.2001; 43:313-23.
  • 19.Menon G, Poskitt EM. Why does congenital heartdisease cause failure to thrive? Arch Dis Child 1985;60: 1134-9.
  • 20.Nydegger A, Bines JE. Energy metabolism ininfants with congenital heart disease. Nutrition2006; 22: 697-704.
  • 21.Rhee EK, Evangelista JK, Nigrin DJ, Erickson LC.Impact of anatomic closure on somatic growthamong small, asymptomatic children withsecundum atrial septal defect. Am J Cardiol 2000;85:1472-5.
  • 22.Çil E, Zincirci M, Bostan OM. Konjenital kalphastalıklarında malnutrisyon prevalansı ve bunuetkileyen faktorler. Turkiye Klinikleri Kardiyoloji2000; 13: 426-33.
  • 23.Cheung MM, Davis AM, Wilkinson JL, WeintraubRG. Long term somatic growth after repair oftetralogy of Fallot: evidence for restoration ofgenetic growth potential. Heart. 2003; 89:1340-3.
  • 24.McElhinney DB, Hedrick HL, Bush DM, et al.Necrotizing enterocolitis in neonates withcongenital heart disease: risk factors and outcomes.Pediatrics 2000; 106:1080-7.
  • 25.Jeffries HE, Wells WJ, Stames VA, Wetzel RC,Moromisato DY. Gastrointestinal morbidity afterNorwood palliation for hypoplastic left heartsyndrome. Ann Thorac Surg 2006; 81:982-7.
Toplam 25 adet kaynakça vardır.

Ayrıntılar

Birincil Dil İngilizce
Konular Sağlık Kurumları Yönetimi, Tıp Eğitimi
Bölüm Original Articles
Yazarlar

Rosa Kasancı Erbay Bu kişi benim

Alper Akın

Kamil Yılmaz

Edip Unal

Mehmet Türe

Duygu Uç

Yayımlanma Tarihi 14 Mart 2025
Gönderilme Tarihi 2 Ekim 2024
Kabul Tarihi 30 Aralık 2024
Yayımlandığı Sayı Yıl 2025 Cilt: 52 Sayı: 1

Kaynak Göster

APA Kasancı Erbay, R., Akın, A., Yılmaz, K., Unal, E., vd. (2025). Prevalence of malnutrition and influencing factors in children with congenital heart disease. Dicle Medical Journal, 52(1), 7-16. https://doi.org/10.5798/dicletip.1657092
AMA Kasancı Erbay R, Akın A, Yılmaz K, Unal E, Türe M, Uç D. Prevalence of malnutrition and influencing factors in children with congenital heart disease. diclemedj. Mart 2025;52(1):7-16. doi:10.5798/dicletip.1657092
Chicago Kasancı Erbay, Rosa, Alper Akın, Kamil Yılmaz, Edip Unal, Mehmet Türe, ve Duygu Uç. “Prevalence of Malnutrition and Influencing Factors in Children With Congenital Heart Disease”. Dicle Medical Journal 52, sy. 1 (Mart 2025): 7-16. https://doi.org/10.5798/dicletip.1657092.
EndNote Kasancı Erbay R, Akın A, Yılmaz K, Unal E, Türe M, Uç D (01 Mart 2025) Prevalence of malnutrition and influencing factors in children with congenital heart disease. Dicle Medical Journal 52 1 7–16.
IEEE R. Kasancı Erbay, A. Akın, K. Yılmaz, E. Unal, M. Türe, ve D. Uç, “Prevalence of malnutrition and influencing factors in children with congenital heart disease”, diclemedj, c. 52, sy. 1, ss. 7–16, 2025, doi: 10.5798/dicletip.1657092.
ISNAD Kasancı Erbay, Rosa vd. “Prevalence of Malnutrition and Influencing Factors in Children With Congenital Heart Disease”. Dicle Medical Journal 52/1 (Mart 2025), 7-16. https://doi.org/10.5798/dicletip.1657092.
JAMA Kasancı Erbay R, Akın A, Yılmaz K, Unal E, Türe M, Uç D. Prevalence of malnutrition and influencing factors in children with congenital heart disease. diclemedj. 2025;52:7–16.
MLA Kasancı Erbay, Rosa vd. “Prevalence of Malnutrition and Influencing Factors in Children With Congenital Heart Disease”. Dicle Medical Journal, c. 52, sy. 1, 2025, ss. 7-16, doi:10.5798/dicletip.1657092.
Vancouver Kasancı Erbay R, Akın A, Yılmaz K, Unal E, Türe M, Uç D. Prevalence of malnutrition and influencing factors in children with congenital heart disease. diclemedj. 2025;52(1):7-16.